MORFOLOGÍA DEL VIRUS DEL DENGUE (INGLÉS)

MORPHOLOGY

• Four serotypes of the dengue virus

• Epidemiology

• Tropicale regions

• Transmitter Aedes aegypti

• 10 million cases annually

• Characteristics

• Is 40 to 60 nm

• Spherical nucleocapsid

• ARN chain

SYMPTOMS

Incubation is five to eight days, Muscle aches It comes scammers, headache, retreorbitrario pain, conjunctival congestion , edemade eyelids, lumbosacral pain , fever punctate macular rash , generalized lymphadenopathy , prostration , anorexia and nausea , slow heartbeat , muscle pain , joint pain , sore throat , rhinitis , changes in taste sensation and some petechiae and ecchymosis

pathogeny

virus replicates in macrophages and monocytes

It causes bone marrow suppression

humural and cellular immune response

It provides protection

The virus replicates in cells of nuclear phagocyte system especially macrophages and monocytes. Pathogenetic mechanisms in cytokines, T lymphocytes, CD4, interlucinas, interferon, tumor necrosis factor and platelet activating factor participate.

suppression occurs in the bone marrow with resulting leukopenia. immune response and later, both humoral and cellular, induce recovery.

pathogeny

virus replicates in macrophages and monocytes

It causes bone marrow suppression

humural and cellular immune response

It provides protection

The virus replicates in cells of nuclear phagocyte system especially macrophages and monocytes. Pathogenetic mechanisms in cytokines, T lymphocytes, CD4, interlucinas, interferon, tumor necrosis factor and platelet activating factor participate.

suppression occurs in the bone marrow with resulting leukopenia. immune response and later, both humoral and cellular, induce recovery

pathogeny

virus replicates in macrophages and monocytes

It causes bone marrow suppression

humural and cellular immune response

It provides protection

The virus replicates in cells of nuclear phagocyte system especially macrophages and monocytes. Pathogenetic mechanisms in cytokines, T lymphocytes, CD4, interlucinas, interferon, tumor necrosis factor and platelet activating factor participate.

suppression occurs in the bone marrow with resulting leukopenia. immune response and later, both humoral and cellular, induce recovery

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